Letter abstract - Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's Disease

By D. Harold, R. Abraham, P. Hollingworth, R. Sims, A. Gerrish, M. Hamshere, Jaspreet Pahwa, V. Moskvina, Kimberley Dowzell, Amy J. Williams and 76 more. 2009

1,585 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/16b95e81d32b942dc70e63c3f4b44df47ccee4f2

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Genetics

1. Christina M. Lill
2. Rudolph E. Tanzi
3. Lars Bertram

In Molecular Biology

1. Rudolph E. Tanzi
2. Fiona M Menzies
3. Christina M. Lill
4. Lars Bertram
5. D. Rubinsztein
6. I. Slaninová
7. N. Mizushima
8. M. Jäättelä

In Physiology

1. Rudolph E. Tanzi
2. Lars Bertram
3. D. Rubinsztein

Citations in the corpus

• Autophagy and Neurodegeneration: Pathogenic Mechanisms and Therapeutic Opportunities. (2017, positive): “Genetic studies have implicated Phosphatidylinositol Binding Clathrin Assembly Protein (PICALM) in Alzheimer’s disease (Harold et al., 2009; Jun et al., 2010) and changes in the level of this protein have been reported in the brains of patients with Alzheimer’s (Ando et al., 2013; Ando et al.,…”
• The genetics of Alzheimer disease. (2012, background): “Genetic association studies remain the most common method for identifying novel LOAD genes and are most often employed in the form of large-scale GWASs that screen the entire human genome for novel disease loci.”