Pathogenesis of liver fibrosis: role of oxidative stress.

By G. Poli. Molecular Aspects of Medicine, 2000

648 citations recorded by the source corpus.

Subfields: Epidemiology, Hepatology

View Paper: https://www.semanticscholar.org/paper/12171e722071916759dcdadf7f826a7edfef909c

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Epidemiology

1. Steven Dooley
2. J. Lefaix
3. Ralf Weiskirchen
4. A. Gressner
5. S. Delanian

In Hepatology

1. Steven Dooley
2. J. Lefaix
3. Ralf Weiskirchen
4. A. Gressner
5. S. Delanian
6. S. Friedman

Citations in the corpus

• Modern pathogenetic concepts of liver fibrosis suggest stellate cells and TGF-β as major players and therapeutic targets (2006, background): “However, non-peptide mediators also are involved in activation and transdifferentiation of HSC since reactive oxygen species (ROS) [26–28], acetaldehyde [29, 30] and lipid mediators [31] can be fibrogenic.”
• Roles of TGF-beta in hepatic fibrosis. (2002, background): “A direct link between TGF-beta signal transduction and TIMP-1 was found in dermal fibroblasts, where a combined cDNA microarray/promoter transactivation approach for the identification of direct TGF-beta target genes was used (85).”
• The radiation-induced fibroatrophic process: therapeutic perspective via the antioxidant pathway. (2004, background): “after its induction by attacks by alcohol, viruses, or silica [68,120].”