Genomic instability in laminopathy-based premature aging
By Baohua Liu, Jianming Wang, K. Chan, W. Tjia, W. Deng, Xinyuan Guan, Jian-Dong Huang, Kai Li, Pui Yin Chau, David J. Chen and 11 more. Nature Medicine, 2005
660 citations recorded by the source corpus.
Subfields: Molecular Biology
View Paper: https://www.semanticscholar.org/paper/11c8c8f2e30401376ed6a80c0f6ad3567f9a30e0
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Molecular Biology
- Linda Partridge
- Manuel Serrano
- Guido Kroemer
- C. López‐Otín
- G. Núñez
- M. Blasco
- L. Galluzzi
- Douglas R. Green
Citations in the corpus
- "Laminopathies": a wide spectrum of human diseases. (2007, background): “Some data suggest that these proteins perturb DNA damage response and repair, resulting in genomic instability [73].”
- The hallmarks of aging. (2013, background): “Nuclear lamins constitute the major components of the nuclear lamina and participate in genome maintenance by providing a scaffold for tethering chromatin and protein complexes that regulate genomic stability (Gonzalez-Suarez et al., 2009; Liu et al., 2005).”
- In Vivo Amelioration of Age-Associated Hallmarks by Partial Reprogramming (2016, positive): “Nuclear lamins maintain proper architecture of the nuclear envelope (Liu et al., 2005).”