Abnormal spine morphology and enhanced LTP in LIMK-1 knockout mice.

By Yanghong Meng, Yu Zhang, Vitali Tregoubov, C. Janus, L. Cruz, Michael B. Jackson, Wei-yang Lu, J. Macdonald, Jay Y. Wang, D. Falls and 1 more. Neuron, 2002

728 citations recorded by the source corpus.

Subfields: Cellular and Molecular Neuroscience, Developmental Neuroscience, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/1100f474f6b862aaf3b60bf60aa3335212a1a90d

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cellular and Molecular Neuroscience

1. T. Tada
2. N. Butcher
3. M. Sheng
4. Ian A. Meinertzhagen
5. Daniel H. Geschwind
6. K. Inokuchi
7. Y. Saitoh
8. Michael D. Ehlers

In Developmental Neuroscience

1. M. Sheng

In Molecular Biology

1. Catherine Lord
2. T. Tada
3. Brian J. O'Roak
4. M. Sheng
5. Danish H Sayed
6. Ian A. Meinertzhagen
7. Shrikant M. Mane
8. Daniel H. Geschwind

Citations in the corpus

• Hippocampal LTP is accompanied by enhanced F-actin content within the dendritic spine that is essential for late LTP maintenance in vivo. (2003, background): “…genetic perturbation of actinPotential Mechanisms by which Reorganized Spine Actin Cytoskeleton Promotes the Late dynamics affects spine structures (Meng et al., 2002; Nakayama et al., 2000; Pak et al., 2001; Penzes et al.,Phase of LTP How can the reorganized spine actin cytoskeleton par- 2001;…”
• Synaptic organization in the adult Drosophila mushroom body calyx (2009, background): “In the honeybee, recurrent GABAergic interneurons innervating the lobes and the calyx (Mobbs, 1982) have been suggested to be involved in olfactory memory formation (Grünewald, 1999).”
• Multiple recurrent de novo CNVs, including duplications of the 7q11.23 Williams syndrome region, are strongly associated with autism. (2011, background): “…several lines of evidence, including atypical deletions (Antonell et al., 2010), mouse models (Fujiwara et al., 2006; Hoogenraad et al., 2002; Meng et al., 2002; Sakurai et al., 2010), and gene expression x phenotype studies (Gao et al., 2010; Korenberg et al., 2000) have already identified…”
• Molecular mechanisms of dendritic spine morphogenesis. (2006, background): “Lim-kinase knockout mice show abnormal spine morphology, abnormal synaptic plasticity and impaired spatial learning [ 40 ].”
• MicroRNAs in development and disease. (2011, background): “In agreement, a knockout model of this protein exhibits a similar phenotype (418).”
• The role of the Rho GTPases in neuronal development. (2005, background): “those found in human WS patients, including heightened locomotor activity, impaired spatial learning, and altered fear responses (Meng et al. 2002).”