Iron and metal regulation in bacteria.
By K. Hantke. Current Opinion in Microbiology, 2001
780 citations recorded by the source corpus.
Subfields: Environmental Engineering, Inorganic Chemistry, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/0fb8a09e020252701e1f879dd19491783dae305c
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In Environmental Engineering
In Inorganic Chemistry
In Nutrition and Dietetics
Citations in the corpus
- Bacterial iron homeostasis. (2003, background): “However, it is now known that the fhuF gene encodes a cytoplasmic ferredoxin-like protein thought to act as a ferrioxamine B reductase in the release of iron from the internalised ferri-siderophore [47,48].”
- Antimicrobial reactive oxygen and nitrogen species: concepts and controversies (2004, background): “pl?P06975">DtxR regulate bacterial iron-uptake systems at the transcriptional leve”
- A common mechanism of cellular death induced by bactericidal antibiotics. (2007, neutral): “For example, further exploration of the posttranscriptional relationship among iron, metabolism, and iron-sulfur cluster-containing proteins (Hantke, 2001) is needed to identify any posttranscriptional events not captured by gene expression analysis that coordinate or trigger this common cell death…”
- Siderophore-Based Iron Acquisition and Pathogen Control (2007, background): “While Fur is the global iron regulator in many gramnegative (e.g., enteric bacteria) and low-GC-content gram-positive (e.g., Bacillus spp.) bacteria, DtxR fulfills a comparable role in gram-positive bacteria with a high GC content (streptomycetes, mycobacteria, and corynebacteria).”
- The impact of iron oxide magnetic nanoparticles on the soil bacterial community (2011, background): “Microorganisms employ various iron uptake systems to secure sufficient supplies from their surroundings (Hantke 2001).”
- The Role of Siderophores in Plant Growth-Promoting Bacteria (2013, background): “In bacteria, gene regulation of siderophore utilization and iron homeostasis is mediated mainly at the transcriptional level by the ferric uptake repressor Fur (in Gram-negative and low mol % GC Gram-positive bacteria) or by the diphtheria toxin regulator DtxR (in Gram-positive high GC contents as s”