Neutrophil swarms require LTB4 and integrins at sites of cell death in vivo
By T. Lämmermann, Philippe V. Afonso, B. Angermann, Ji Ming Wang, W. Kastenmüller, C. Parent, Ronald N. Germain. Nature, 2013
876 citations recorded by the source corpus.
Subfields: Immunology, Immunology and Allergy
View Paper: https://www.semanticscholar.org/paper/0f9aaecca99c9dcf8a19116ed406b25c5a39dcd9
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In Immunology
In Immunology and Allergy
Citations in the corpus
- Pro-Resolving lipid mediators and Mechanisms in the resolution of acute inflammation (2014, background): “…post cecal ligation and puncture (CLP) in a model of sepsis appears to have little effect on vascular tone and permeability, but may have blunted the innate immune response (specifically neutrophil trafficking), exacerbating the infective insult (Lammermann et al., 2013; Rios-Santos et al., 2003).”
- Cellular and molecular mechanisms of skin wound healing (2024, neutral): “Activated neutrophils also trigger a positive feedback response by releasing autocrine and paracrine signals, leading to swarming behaviour by other neutrophils 84,85 .”
- The chemokine system in innate immunity. (2015, neutral): “2C) (Lammermann et al. 2013).”
- DAMPs, PAMPs, and LAMPs in Immunity and Sterile Inflammation. (2020, background): “Indeed, neutrophils tend to migrate in an exponentially clustered fashion through the interstitial space, a process also referred to as neutrophil swarming (63, 65, 66).”