Human Coronavirus OC43 Associated with Fatal Encephalitis.
By Sofia Morfopoulou, Julianne R. Brown, E. Davies, G. Anderson, A. Virasami, W. Qasim, W. Chong, M. Hubank, V. Plagnol, M. Desforges and 3 more. 2016
259 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Infectious Diseases
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Infectious Diseases
- Kwok-Yung Yuen
- J. Chan
- K. To
- Kwok-Hung Chan
- Shuofeng Yuan
- Christian Drosten
- H. Tsoi
- Susanna K. P. Lau
Citations in the corpus
- Human coronaviruses OC43 and HKU1 bind to 9-O-acetylated sialic acids via a conserved receptor-binding site in spike protein domain A (2019, background): “Although generally associated with common colds, HKU1 and OC43 may cause severe and sometimes fatal pulmonary infections in the frail (7, 8), and in rare instances, OC43 may cause lethal encephalitis (9).”
- Comparative tropism, replication kinetics, and cell damage profiling of SARS-CoV-2 and SARS-CoV with implications for clinical manifestations, transmissibility, and laboratory studies of COVID-19: an observational study (2020, result): “25 Our data also provide new insights into the apparently high transmissibility of SARS-CoV-2.”
- Hosts and Sources of Endemic Human Coronaviruses (2018, background): “In particular, HCoV-OC43 has been suspected to play a role in neurological diseases such as chronic demyelinating disease and acute encephalomyelitis (Morfopoulou et al., 2016; Murray et al., 1992; Yeh et al., 2004) .”