Mitochondria and Mitophagy: The Yin and Yang of Cell Death Control
By Dieter A. Kubli, Åsa B. Gustafsson. Circulation Research, 2012
770 citations recorded by the source corpus.
Subfields: Epidemiology, Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/0e8145879427b8381e7fd5851fd17f0adc23f9a1
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Epidemiology
In Molecular Biology
- Paul D. Thomas
- Huaiyu Mi
- Pascale Gaudet
- Anushya Muruganujan
- Suzanna Lewis
- Marc Feuermann
- Dustin Ebert
- Gene Ontology Consortium
Citations in the corpus
- Large-scale inference of gene function through phylogenetic annotation of Gene Ontology terms: case study of the apoptosis and autophagy cellular processes (2016, background): “Depending on the cellular component targeted for recycling, the macroautophagy process is known as mitophagy for mitochondria degradation (20, 21), nucleophagy for nuclei degradation (22), pexophagy for peroxisomes breakdown (12, 23), riboph-agy for ribosomes (24), reticulophagy for endoplasmic…”
- Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets (2015, background): “As described earlier, mitochondria are responsible for providing cardiomyocytes with a continuous supply of ATP, but also participate in regulating cell death due to the production of ROS (Kubli and Gustafsson 2012).”
- Ischemia/Reperfusion (2016, neutral): “lihood of I/R-induced cell death in preconditioned tissues (283,327,457).”