S-Glutathiolation by peroxynitrite activates SERCA during arterial relaxation by nitric oxide

By T. Adachi, R. Weisbrod, D. Pimentel, Jia Ying, V. Sharov, C. Schöneich, R. Cohen. Nature Medicine, 2004

658 citations recorded by the source corpus.

Subfields: Biochemistry, Molecular Biology, Physiology

View Paper: https://www.semanticscholar.org/paper/0dbbabad224153e70a773acbad28c5eab83533e4

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In Molecular Biology

1. K. Krause
2. K. Bedard
3. C. Kevil
4. S. Bir
5. G. Kolluru

In Physiology

1. K. Krause
2. K. Bedard
3. K. Griendling

Citations in the corpus

• The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology. (2007, background): “ROS modulate the activity of Ca(2) ATPase pumps (10, 316, 734) in a bimodal fashion.”
• Endothelial Dysfunction and Diabetes: Effects on Angiogenesis, Vascular Remodeling, and Wound Healing (2012, background): “Nitric oxide (NO) stimulates SERCA to decrease intracellular Ca2+ thereby allowing relaxation of cardiac, skeletal, and vascular smooth muscle [205].”
• Biological markers of oxidative stress: Applications to cardiovascular research and practice (2013, background): “Schematic illustration illustrating the functional effect of glutathionylation of key cardiovascular proteins eNOS [58], SERCA [13], and Na þ –”