Characterization of an ocular photopigment capable of driving pupillary constriction in mice
By R. Lucas, R. Douglas, R. Foster. Nature Neuroscience, 2001
631 citations recorded by the source corpus.
Subfields: Cellular and Molecular Neuroscience, Endocrine and Autonomic Systems, Sensory Systems
View Paper: https://www.semanticscholar.org/paper/0af27efe1276e617003ed51872653dcbc10d99fd
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In Cellular and Molecular Neuroscience
In Endocrine and Autonomic Systems
In Sensory Systems
Citations in the corpus
- Migraine pathophysiology: anatomy of the trigeminovascular pathway and associated neurological symptoms, CSD, sensitization and modulation of pain (2013, background): “The first pathway allows the formation of images by photoactivation of rods and cones, and the second allows regulation of biological functions such as circadian photoentrainment, pupillary reflex, and melatonin release by activation of intrinsically photosensitive retinal ganglion cells containing ”
- Central projections of melanopsin‐expressing retinal ganglion cells in the mouse (2006, background): “Together, these data support the view that inputs from ganglion-cell photoreceptors sustain photic effects on circadian phase and pupil diameter in mice lacking functional rods and cones (Yoshimura and Ebihara, 1996; Lucas et al., 2001; Hattar et al., 2003).”
- An action spectrum for melatonin suppression: evidence for a novel non‐rod, non‐cone photoreceptor system in humans (2001, background): “Recent studies using rodless/coneless mice have shown that novel non-rod, non-cone photoreceptor(s) may mediate circadian photoentrainment (Freedman et al. 1999), acute light responses (melatonin suppression) (Lucas et al. 1999) and pupil constriction (Lucas et al. 2001).”
- Neural remodeling in retinal degeneration. (2003, methodology): “What transpires in the remnant neural retina of humans and animals deprived of a sensory retina? There is some evidence that circadian signaling and even pupillary responses can be preserved by non-traditional photopigment signaling elements that drive subcortical pathways (Freedman et al., 1999; Lu”
- Physiology of circadian entrainment. (2010, background): “Since the presence of melanopsin (193) and chryptochromes (463) in human ganglion cells, as well as a decrease of PLR in glaucomatous patients had been demonstrated (66), one might conjecture that individuals with severe retinal ganglion cell loss (i.e., glaucoma) would exhibit circadian rhythm desy”