Proinflammatory cytokine synthesis in the injured mouse spinal cord: Multiphasic expression pattern and identification of the cell types involved
By Isabelle Pineau, S. Lacroix. The Journal of comparative neurology, 2007
601 citations recorded by the source corpus.
Subfields: Developmental Neuroscience, Neurology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/0a454930f07b2f9d16b83402baecb9dfd34cf76e
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In Neurology
In Pathology and Forensic Medicine
Citations in the corpus
- TNF and increased intracellular iron alter macrophage polarization to a detrimental M1 phenotype in the injured spinal cord. (2014, unknown): “Our search for potential candidates that can influence M1 polarization focused on TNF as TNF mRNA is rapidly upregulated after contusion injury in mice and is expressed by microglia and other CNS cell types (Pineau and Lacroix, 2007).”
- Traumatic Spinal Cord Injury: An Overview of Pathophysiology, Models and Acute Injury Mechanisms (2019, background): “Within minutes of injury, production of IL-1β is significantly elevated in astrocytes and microglia (203).”
- Repertoire of microglial and macrophage responses after spinal cord injury (2011, background): “Microglia express the mRNA for IL-1β within 1–12 hours after spinal cord contusion injur”
- Inflammation and its role in neuroprotection, axonal regeneration and functional recovery after spinal cord injury. (2008, unknown): “Curves were generated using data from the following references: A) (11;67;169); B) (81;132;136;159;188): C) (129;163;180); D) (24; 59;72;108); E) (120;133;179).”