Nutritional, dietary and postprandial oxidative stress.

By H. Sies, W. Stahl, A. Sevanian. Journal of NutriLife, 2005

601 citations recorded by the source corpus.

Subfields: Biochemistry, Organic Chemistry

View Paper: https://www.semanticscholar.org/paper/0a0ae3497557146994ef19a9a789b8167157b314

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Citations in the corpus

• Oxidative stress and metabolic disorders: Pathogenesis and therapeutic strategies. (2016, background): “high-carbohydrate diet and continuous hyper nutrition can cause increased oxidative stress through activation of intracellular pathways such as NOX, oxidative phosphorylation in mitochondria, glycoxidation, protein kinase C (PKC) and polyol pathway [59-62].”
• Biochemistry of Free Radicals and Antioxidants (2010, neutral): “Some antioxidants can interact with other antioxidants regenerating their original properties; this mechanism is often referred to as the “antioxidant network” [38].”
• Studies on free radicals, antioxidants, and co-factors (2007, background): “A high level of oxidative stress can induce apoptosis or even necrosis; however, a low level of oxidative stress can stimulate cell division and thus promote tumor growth (Dreher and Junod 1996).”