Heme oxygenase-1/carbon monoxide: from basic science to therapeutic applications.
By S. Ryter, J. Alam, A. Choi. Physiological Reviews, 2006
2,330 citations recorded by the source corpus.
Subfields: Molecular Biology, Pediatrics, Perinatology and Child Health, Pharmacology
View Paper: https://www.semanticscholar.org/paper/09dce9d7c5d1253ee7d88388a181460108e3b2af
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In Molecular Biology
In Pediatrics, Perinatology and Child Health
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Citations in the corpus
- Oxidative stress: an essential factor in the pathogenesis of gastrointestinal mucosal diseases. (2014, background): “Two distinct HO isoforms, HO-1 and HO-2, have been reported (253).”
- The therapeutic potential of carbon monoxide (2010, background): “Contrary to the common perception that CO is a substance that causes harm for mammalian organisms that rely on oxygen transport and mitochondrial respiration, the physiological roles attributed to endogenously generated CO, as well as the beneficial effects exerted by low doses of applied CO gas, ar”
- The Role of Macrophages in Acute and Chronic Wound Healing and Interventions to Promote Pro-wound Healing Phenotypes (2018, background): “Carbon monoxide and bilirubin can exert anti-inflammatory properties to help wound healing (Kapitulnik, 2004; Ryter et al., 2006) however, differential regulation of the iron product can promote M1- or M2like macrophages (Cairo et al., 2011).”
- Carbon monoxide intoxication: an updated review. (2007, background): “There are also endogenous sources of CO, such as during the heme degradation to bile pigments, catalyzed by heme oxygenases [4].”
- Cell Biology of Ischemia/Reperfusion Injury (2012, neutral): “This action prevents heme-mediated oxidative injury (Bauer and Bauer, 2002; Devey et al., 2009; Ellett et al., 2010; Ryter et al., 2006; Stocker et al., 1987; Suematsu and Ishimura, 2000; Tomiyama et al., 2008).”
- Ischemia/Reperfusion (2016, neutral): “enger receptor, which is then followed by heme oxygenase-1 (HO-1)-dependent heme degradation (54, 55, 190, 211, 264, 274, 432, 452, 677, 751, 787).”
- Role of Kupffer cells in host defense and liver disease (2006, background): “Rather, the rapid elimination of bacteria taken up by the liver is dependent on the complex interaction of KC and microbicidal neutrophils, which immigrate rapidly in response to infection (28, 29, 58, 59).”
- Physiological implications of hydrogen sulfide: a whiff exploration that blossomed. (2012, background): “H2S-induced upregulation of HO expression in pulmonary smooth muscle cells (451) and macrophages (501) has been shown, which is believed as the consequence of ERK activation (533).”