Toll-like receptors and innate immunity.

By Himanshu Kumar, Taro Kawai, Shizuo Akira. Biochemical and Biophysical Research Communications - BBRC, 2009

704 citations recorded by the source corpus.

Subfields: Epidemiology, Immunology

View Paper: https://www.semanticscholar.org/paper/09a49543a72df6382e36ad0f87383a01b1771b8f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Immunology

1. I. Fraser
2. Michael G. Dorrington

Citations in the corpus

• NF-κB Signaling in Macrophages: Dynamics, Crosstalk, and Signal Integration (2019, background): “TLR4 signaling through the adaptor molecule MyD88 leads to the activation of NF-κB, mitogenactivated protein kinase (MAPK), and IRF5 pathways (30), while subsequent endosomal TLR4 signaling through TIR-domaincontaining adaptor protein inducing interferon-β (TRIF) leads to the activation of IRF3, and”
• INFLAMMATION AND LIPID SIGNALING IN THE ETIOLOGY OF INSULIN RESISTANCE (2012, neutral): “Tlr2 and Tlr4 are members of the Tlr family of receptors that recognize pathogen-associated molecular patterns (PAMPs), as well as endogenously derived indicators of tissue injury (Kumar et al., 2009).”