Paraneoplastic limbic encephalitis: neurological symptoms, immunological findings and tumour association in 50 patients.

By S. Gultekin, Myrna R. Rosenfeld, R. Voltz, J. Eichen, J. Posner, J. Dalmau. Brain : a journal of neurology, 2000

1,131 citations recorded by the source corpus.

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In Neurology

J. Dalmau

Citations in the corpus

Clinical analysis of anti-Ma2-associated encephalitis. (2004, background): “Neurological symptoms were coded into the following syndromes: limbic encephalitis (Gultekin et al., 2000), diencephalic encephalitis, brainstem encephalitis and other.”
Spectrum of neurological syndromes associated with glutamic acid decarboxylase antibodies: diagnostic clues for this association. (2008, background): “In two of them, the brain MRI was normal, a feature that does not rule out this diagnosis (Gultekin et al., 2000), and both presented positive IS of GAD-ab.”
Paraneoplastic anti–N‐methyl‐D‐aspartate receptor encephalitis associated with ovarian teratoma (2007, background): “One cause of autoimmune encephalitis is the paraneoplastic manifestation of a neoplasm.(1) Until now, most paraneoplastic encephalitides have been associated with antibodies to intracellular onconeuronal proteins and cytotoxic T cells presumably against the same proteins.”
AMPA receptor antibodies in limbic encephalitis alter synaptic receptor location (2009, neutral): “Until recently, most cases of LE were thought to be paraneoplastic, mediated by immune responses against intracellular antigens, and poorly responsive to treatment.(2) However, the clinical and immunological spectra of LE are far more extensive than initially considered, and recent studies demonstra”
Recommended diagnostic criteria for paraneoplastic neurological syndromes (2004, background): “11 The presence of high levels of voltage gated potassium channel (VGKC) antibodies may suggest the diagnosis of idiopathic limbic encephalitis 12 but these antibodies are also found in a few patients with a paraneoplastic origin, 13 so the test should not be used to exclude a paraneoplastic cause…”
Potassium channel antibody-associated encephalopathy: a potentially immunotherapy-responsive form of limbic encephalitis. (2004, neutral): “Clinically, these cases do not differ substantially from other forms of amnesic encephalopathies, such as the paraneoplastic form of limbic encephalitis associated with small-cell lung cancer and Hu antibodies (Gultekin et al., 2000), except in the relative absence of cerebellar and brainstem involv”