Oxygen, oxidative stress, hypoxia, and heart failure.

By F. Giordano. Journal of Clinical Investigation, 2005

1,495 citations recorded by the source corpus.

Subfields: Molecular Biology, Pathology and Forensic Medicine, Physiology

View Paper: https://www.semanticscholar.org/paper/01eaba8b7550f369609bbed4caf38f98238eadf3

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

1. Y. Tham
2. J. Ooi
3. Bianca C. Bernardo
4. K. Weeks
5. J. McMullen

Citations in the corpus

• NADPH Oxidases in Cardiovascular Health and Disease (2006, neutral): “Ang II increases VSMC growth and hypertrophy and this process is dependent on NADPH oxidase-derived ROS, probably mainly Nox1-derived (370).”
• Pathophysiology of cardiac hypertrophy and heart failure: signaling pathways and novel therapeutic targets (2015, background): “Under normal physiological conditions, ROS act as mediators to induce adaptive responses in the heart (Song et al. 2014), and the formation of excessive ROS is prevented by intrinsic antioxidant systems within the cell (Giordano 2005).”