Pathogenesis and clinical features of psoriasis.
By C. Griffiths, J. Barker. The Lancet, 2007
1,936 citations recorded by the source corpus.
Subfields: Dermatology, Immunology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/01e74323df3a4da3833738f42f6df14953cb819d
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In Dermatology
In Immunology
Citations in the corpus
- The risk of depression, anxiety and suicidality in patients with psoriasis: A population-based cohort study (2010, background): “tors and results in chronic T helper (TH)1 and TH17 inflammation in the skin, blood, and in some patients, joints.(5,6)”
- The Role of Tumor Necrosis Factor Alpha (TNF-α) in Autoimmune Disease and Current TNF-α Inhibitors in Therapeutics (2021, background): “The lesions of PS reflect the epidermal hyperplasia, angiogenesis, and inflammatory infiltration of leukocytes into the dermis as a result of the dysregulation of skin immune responses [48].”
- Obesity in autoimmune diseases: not a passive bystander. (2014, background): “The pathogenesis of both psoriasis and PsA is thought to result from the interplay between a strong genetic background, environmental factors, and immune dysfunction [267].”
- Efalizumab binding to the LFA-1 αL I domain blocks ICAM-1 binding via steric hindrance (2009, background): “Psoriasis is a very common skin disease that is characterized by red or salmon pink color, white or silver scaly and raised plaques (22).”
- Global epidemiology of psoriasis: a systematic review of incidence and prevalence. (2013, background): “Psoriasis is associated with cardiovascular disease, depressive illness, and psoriatic arthritis (Griffiths and Barker, 2007).”
- Psoriasis and systemic inflammatory diseases: potential mechanistic links between skin disease and co-morbid conditions. (2010, background): “VEGF levels are increased in plaques of psoriasis and serum concentration of VEGF correlates with clinical severity of disease (Griffiths and Barker, 2007).”