Jonathan H. Lin

Cell Biology · 2007–2009

A researcher in the Science corpus: 3 papers; trusts 3 researchers and is trusted by 2 researchers here, as inferred from the citation graph.

Subfields

• Cell Biology — 56 citations into it; #1,295 of 2,073 by trust here, #629 by citations
• Molecular Biology — 40 citations into it; #13,153 of 17,773 by trust here, #7,747 by citations
• Epidemiology — 22 citations into it; #2,092 of 2,148 by trust here, #998 by citations
• Surgery — 18 citations into it

Whom Jonathan H. Lin trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

1. H. Harding
2. D. Ron
3. Ann-Hwee Lee
4. Hiderou Yoshida
5. N. Iwakoshi
6. I. Novoa
7. Yuhong Zhang
8. J. Hollien
9. Huiqing Zeng
10. S. Oyadomari

Who trusts Jonathan H. Lin overall

1. C. Hetz
2. G. Hotamışlıgil
3. J. Cubillos-Ruiz
4. F. Papa
5. A. Kaser
6. R. Kaufman
7. Laurie H. Glimcher
8. Sarah Bettigole

Papers from Jonathan H. Lin's perspective

Ordered by how strongly the researchers Jonathan H. Lin trusts endorse each paper. Same corpus, a different order for every researcher.

1. Regulated translation initiation controls stress-induced gene expression in mammalian cells. (Molecules and Cells)
2. Translational control is required for the unfolded protein response and in vivo glucose homeostasis. (Molecules and Cells)
3. An integrated stress response regulates amino acid metabolism and resistance to oxidative stress. (Molecules and Cells)
4. IRE1-mediated unconventional mRNA splicing and S2P-mediated ATF6 cleavage merge to regulate XBP1 in signaling the unfolded protein response. (Genes & Development)
5. The PERK Eukaryotic Initiation Factor 2α Kinase Is Required for the Development of the Skeletal System, Postnatal Growth, and the Function and Viability of the Pancreas (Molecular and Cellular Biology)
6. Diabetes mellitus and exocrine pancreatic dysfunction in perk-/- mice reveals a role for translational control in secretory cell survival. (Molecules and Cells)
7. Proapoptotic BAX and BAK: A Requisite Gateway to Mitochondrial Dysfunction and Death (Science)
8. A time-dependent phase shift in the mammalian unfolded protein response. (Developmental Cell)
9. ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats. (Genes & Development)
10. Bax and Bak can localize to the endoplasmic reticulum to initiate apoptosis (Journal of Cell Biology)

Their papers

• IRE1 Signaling Affects Cell Fate During the Unfolded Protein Response (2007) — 1,392 citations
• Regulated Ire1-dependent decay of messenger RNAs in mammalian cells (2009) — 1,010 citations
• Endoplasmic reticulum stress in disease pathogenesis. (2008) — 819 citations