Mary Y. Wu

Molecular Biology · 2008–2009

A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 1 researcher here, as inferred from the citation graph.

Subfields

Whom Mary Y. Wu trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. C. Hill
  2. J. Massagué
  3. P. ten Dijke

Who trusts Mary Y. Wu overall

  1. David M. Sabatini

Papers from Mary Y. Wu's perspective

Ordered by how strongly the researchers Mary Y. Wu trusts endorse each paper. Same corpus, a different order for every researcher.

  1. TSC2 mediates cellular energy response to control cell growth and survival. (Cell)
  2. TOR signaling in growth and metabolism. (Cell)
  3. S6K1−/−/S6K2−/− Mice Exhibit Perinatal Lethality and Rapamycin-Sensitive 5′-Terminal Oligopyrimidine mRNA Translation and Reveal a Mitogen-Activated Protein Kinase-Dependent S6 Kinase Pathway (Molecular and Cellular Biology)
  4. Prolonged rapamycin treatment inhibits mTORC2 assembly and Akt/PKB. (Molecules and Cells)
  5. TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
  6. Akt maintains cell size and survival by increasing mTOR-dependent nutrient uptake. (Molecular Biology of the Cell)
  7. Insulin signalling to mTOR mediated by the Akt/PKB substrate PRAS40 (Nature Cell Biology)
  8. Ablation in mice of the mTORC components raptor, rictor, or mLST8 reveals that mTORC2 is required for signaling to Akt-FOXO and PKCalpha, but not S6K1. (Developmental Cell)
  9. Akt regulates growth by directly phosphorylating Tsc2 (Nature Cell Biology)
  10. Growing roles for the mTOR pathway. (Current Opinion in Cell Biology)

Their papers