M. S. Woo
Materials Chemistry · 2007–2009
A researcher in the Science corpus: 4 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Materials Chemistry — 5 citations into it
Whom M. S. Woo trusts overall
Their strongest outbound trust, strongest first, inferred from their citations.
Papers from M. S. Woo's perspective
Ordered by how strongly the researchers M. S. Woo trusts endorse each paper. Same corpus, a different order for every researcher.
- Improved methods for building protein models in electron density maps and the location of errors in these models.
- Gefitinib-Sensitizing EGFR Mutations in Lung Cancer Activate Anti-Apoptotic Pathways (Science)
- Refinement of macromolecular structures by the maximum-likelihood method.
- Untangling the ErbB signalling network (Nature reviews. Molecular cell biology)
- EGFR Mutations in Lung Cancer: Correlation with Clinical Response to Gefitinib Therapy (Science)
- Somatic mutations of epidermal growth factor receptor signaling pathway in lung cancers (International Journal of Cancer)
- Structure of the Epidermal Growth Factor Receptor Kinase Domain Alone and in Complex with a 4-Anilinoquinazoline Inhibitor* (Journal of Biological Chemistry)
- Acquired Resistance of Lung Adenocarcinomas to Gefitinib or Erlotinib Is Associated with a Second Mutation in the EGFR Kinase Domain (PLoS Medicine)
- ZD1839 (Iressa): an orally active inhibitor of epidermal growth factor signaling with potential for cancer therapy. (Cancer Research)
- ERBB Receptors and Cancer: The Complexity of Targeted Inhibitors (Nature Reviews. Cancer)
Their papers
- Characterizing the cancer genome in lung adenocarcinoma (2007) — 1,146 citations
- SOX2 Is an Amplified Lineage Survival Oncogene in Lung and Esophageal Squamous Cell Carcinomas (2009) — 956 citations
- Structures of lung cancer-derived EGFR mutants and inhibitor complexes: mechanism of activation and insights into differential inhibitor sensitivity. (2007) — 1,098 citations
- The T790M mutation in EGFR kinase causes drug resistance by increasing the affinity for ATP (2008) — 2,013 citations