E. Junn

Neurology · 2013

A researcher in the Science corpus: 1 paper; trusts 3 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom E. Junn trusts overall

Their strongest outbound trust, strongest first, inferred from their citations.

  1. A. Schapira

Papers from E. Junn's perspective

Ordered by how strongly the researchers E. Junn trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin (Nature)
  2. Parkin is recruited selectively to impaired mitochondria and promotes their autophagy (Journal of Cell Biology)
  3. Mitochondria in the aetiology and pathogenesis of Parkinson's disease. (Lancet Neurology)
  4. Mitochondrial Dysfunction and Oxidative Damage in parkin-deficient Mice* (Journal of Biological Chemistry)
  5. Mitochondrial pathology and muscle and dopaminergic neuron degeneration caused by inactivation of Drosophila Pink1 is rescued by Parkin. (Proceedings of the National Academy of Sciences of the United States of America)
  6. Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants (Proceedings of the National Academy of Sciences of the United States of America)
  7. PINK1 Is Selectively Stabilized on Impaired Mitochondria to Activate Parkin (PLoS Biology)
  8. Loss of PINK1 Function Promotes Mitophagy through Effects on Oxidative Stress and Mitochondrial Fission* (Journal of Biological Chemistry)
  9. Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Nature)
  10. Hereditary Early-Onset Parkinson's Disease Caused by Mutations in PINK1 (Science)

Their papers