E. B. Butler

Molecular Biology · 2013

A researcher in the Science corpus: 1 paper; trusts 2 researchers and is trusted by 0 researchers here, as inferred from the citation graph.

Subfields

Whom E. B. Butler trusts

Their strongest outbound trust, strongest first, inferred from their citations.

  1. Craig B. Thompson (in Molecular Biology)
  2. C. V. Dang (in Molecular Biology)

Papers from E. B. Butler's perspective

Ordered by how strongly the researchers E. B. Butler trusts endorse each paper. Same corpus, a different order for every researcher.

  1. Why do cancers have high aerobic glycolysis? (Nature Reviews. Cancer)
  2. Attenuation of LDH-A expression uncovers a link between glycolysis, mitochondrial physiology, and tumor maintenance. (Cancer Cell)
  3. Beyond aerobic glycolysis: Transformed cells can engage in glutamine metabolism that exceeds the requirement for protein and nucleotide synthesis (Proceedings of the National Academy of Sciences of the United States of America)
  4. The M2 splice isoform of pyruvate kinase is important for cancer metabolism and tumour growth (Nature)
  5. Brick by brick: metabolism and tumor cell growth. (Current Opinion in Genetics and Development)
  6. Targeting mitochondrial glutaminase activity inhibits oncogenic transformation (Cancer Cell)
  7. Inhibition of lactate dehydrogenase A induces oxidative stress and inhibits tumor progression (Proceedings of the National Academy of Sciences of the United States of America)
  8. Understanding the Warburg Effect: The Metabolic Requirements of Cell Proliferation (Science)
  9. Dichloroacetate (DCA) as a potential metabolic-targeting therapy for cancer (British Journal of Cancer)
  10. Hypoxia promotes invasive growth by transcriptional activation of the met protooncogene. (Cancer Cell)

Their papers