A. D’alessio
Molecular Biology · with Bellacosa · 2002–2012
A researcher in the Science corpus: 2 papers; trusts 1 researcher and is trusted by 0 researchers here, as inferred from the citation graph.
Subfields
- Molecular Biology — 29 citations into it
Whom A. D’alessio trusts
Their strongest outbound trust, strongest first, inferred from their citations.
- Lewis C. Cantley (in Molecular Biology)
Papers from A. D’alessio's perspective
Ordered by how strongly the researchers A. D’alessio trusts endorse each paper. Same corpus, a different order for every researcher.
- Mutations of the BRAF gene in human cancer (Nature)
- TSC2 is phosphorylated and inhibited by Akt and suppresses mTOR signalling (Nature Cell Biology)
- The evolution of phosphatidylinositol 3-kinases as regulators of growth and metabolism (Nature reviews genetics)
- The TSC1-2 tumor suppressor controls insulin–PI3K signaling via regulation of IRS proteins (Journal of Cell Biology)
- Ras, PI(3)K and mTOR signalling controls tumour cell growth (Nature)
- Tumor-promoting phorbol esters and activated Ras inactivate the tuberous sclerosis tumor suppressor complex via p90 ribosomal S6 kinase. (Proceedings of the National Academy of Sciences of the United States of America)
- Inhibition of mTORC1 leads to MAPK pathway activation through a PI3K-dependent feedback loop in human cancer. (Journal of Clinical Investigation)
- Targeting RAS signalling pathways in cancer therapy (Nature Reviews. Cancer)
- PI3K pathway alterations in cancer: variations on a theme (Oncogene)
- AKT inhibition relieves feedback suppression of receptor tyrosine kinase expression and activity (Cancer Cell)
Their papers
- Cytoplasmic relocalization and inhibition of the cyclin-dependent kinase inhibitor p27Kip1 by PKB/Akt-mediated phosphorylation in breast cancer (2002) — 772 citations
- The RAS/RAF/MEK/ERK and the PI3K/AKT signalling pathways: role in cancer pathogenesis and implications for therapeutic approaches (2012) — 711 citations