Mechanical stress activates angiotensin II type 1 receptor without the involvement of angiotensin II

By Y. Zou, H. Akazawa, Yingjie Qin, Masanori Sano, H. Takano, T. Minamino, N. Makita, K. Iwanaga, Weidong Zhu, S. Kudoh and 9 more. Nature Cell Biology, 2004

687 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/ff415e7964e043d84dc8798cd105c337873ea5cf

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Cardiology and Cardiovascular Medicine

1. Stanley Nattel
2. J. Heineke
3. J. Molkentin
4. Nikolaos G. Frangogiannis

In Molecular Biology

1. J. Heineke
2. J. Molkentin
3. Nikolaos G. Frangogiannis

Citations in the corpus

• Cardiac fibrosis. (2020, background): “ways require angiotensin II stimulation but may be directly activated by mechanical stress, stimulating a hypertrophic response.(95) Whether”
• Atrial fibrosis: mechanisms and clinical relevance in atrial fibrillation. (2008, background): “Mechanical stretch induces cardiomyocyte itogen-activated protein kinase signaling through direct ctivation of AT1 receptors (84).”
• Regulation of cardiac hypertrophy by intracellular signalling pathways (2006, background): “This leads to the activation of the extracellular signal-regulated kinase (ERK) and the induction of hypertroph”