Immunologic aspects of chronic obstructive pulmonary disease.
By M. Cosio, M. Saetta, Alvar Agusti. New England Journal of Medicine, 2009
831 citations recorded by the source corpus.
Subfields: Emergency Medical Services, Physiology, Pulmonary and Respiratory Medicine
View Paper: https://www.semanticscholar.org/paper/ff026d71eef5d6847a56af1ae03e710273a88282
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Emergency Medical Services
In Physiology
In Pulmonary and Respiratory Medicine
- P. Barnes
- R. Rodríguez-Roisín
- C. Jenkins
- Sonia Buist
- Jan Zieliński
- Yoshinosuke Fukuchi
- S. Hurd
- A. Anzueto
Citations in the corpus
- IL‐12 and IL‐7 synergize to control mucosal‐associated invariant T‐cell cytotoxic responses to bacterial infection (2017, background): “Microbial dysbiosis in COPD is dominated by a loss of microbial 94 diversity and overgrowth of key species, with NTHi-induced exacerbations of COPD being 95 the main drivers of disease progression and mortality [7][11][18][19].”
- Global strategy for the diagnosis, management, and prevention of chronic obstructive pulmonary disease: GOLD executive summary. (2007, background): “Lung inflammation persists after smoking cessation through unknown mechanisms, although autoantigens and persistent microorganisms may play a role (36).”
- Editorial: Global Initiative for Chronic Obstructive Lung Disease (GOLD) 2023 Guidelines for COPD, Including COVID-19, Climate Change, and Air Pollution (2023, neutral): “Воспалительный процесс в легких продолжается и после прекращения курения, механизмы этого эффекта пока неизвестны, но обсуждается роль аутоиммунных процессов и персисти рующей инфекции [87].”