Multiorgan and Renal Tropism of SARS-CoV-2
By Victor G. Puelles, M. Lütgehetmann, M. Lindenmeyer, J. Sperhake, M. N. Wong, L. Allweiss, S. Chilla, A. Heinemann, Nicola Wanner, Shuya Liu and 13 more. New England Journal of Medicine, 2020
1,647 citations recorded by the source corpus.
Multiorgan and Renal Tropism of SARS-CoV-2 In this autopsy series, the authors found that SARS-CoV-2 has an organotropism beyond the respiratory tract, including the kidneys, heart, liver, and brai...
Subfields: Dermatology, Infectious Diseases, Neurology
View Paper: https://www.semanticscholar.org/paper/fe07862535acd26c505aaa2e1003e3e45f70bdcb
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- Yiyin Chen
- S. Klein
- B. Garibaldi
- Huifen Li
- Cun-jin Wu
- Nicole M. Osevala
- J. Margolick
- G. Pawelec
Citations in the corpus
- COVID-19: angiotensin-converting enzyme 2 (ACE2) expression and tissue susceptibility to SARS-CoV-2 infection (2021, methodology): “Pre-existing comorbidities may also be directly associated to higher rates of SARS-CoV-2 organ tropism, as demonstrated in post-mortem analyses of the lungs, pharynx, kidneys, liver, and heart [35].”
- Aging in COVID-19: Vulnerability, immunity and intervention (2020, positive): “While SARS-CoV-2 has been characterized regarding its neurotrophic and neuroinvasive properties (Baig et al., 2020; Puelles et al., 2020), inflammatory cytokine release and immunopathology caused by this”