Atherothrombosis and high-risk plaque: part I: evolving concepts.

By Valentin Fuster, P. Moreno, Z. Fayad, R. Corti, J. Badimón. Journal of the American College of Cardiology, 2005

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Management of acute myocardial infarction in patients presenting with persistent ST-segment elevation: the Task Force on the Management of ST-Segment Elevation Acute Myocardial Infarction of the European Society of Cardiology. (2008, background): “The risk of plaque disruption depends on plaque composition and vulnerability (plaque type) and degree of stenosis (plaque size).(3) As many as three-quarters of all infarct-related thrombi appear to evolve over plaques causing only mild to moderate stenosis.”
Myocardial no-reflow in humans. (2009, background): “consequences of coronary ligation of a nonatherosclerotic coronary artery, however, cannot be directly extrapolated to the human situation where myocardial infarction is caused by occlusive coronary thrombosis superimposed onto an atherosclerotic unstable plaque (13).”