Full haplotype-mismatched hematopoietic stem-cell transplantation: a phase II study in patients with acute leukemia at high risk of relapse.

By F. Aversa, A. Terenzi, A. Tabilio, F. Falzetti, A. Carotti, S. Ballanti, R. Felicini, F. Falcinelli, A. Velardi, L. Ruggeri and 8 more. Journal of Clinical Oncology, 2005

714 citations recorded by the source corpus.

Subfields: Hematology, Public Health, Environmental and Occupational Health

View Paper: https://www.semanticscholar.org/paper/f79cc2a0287d6bbce5500c833cd4ad19cda2ebdf

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M. Di Ianni
L. Ruggeri
F. Falzetti
L. Luznik
A. Carotti
Antonella Mancusi
F. Aversa
A. Terenzi

Citations in the corpus

Tregs prevent GVHD and promote immune reconstitution in HLA-haploidentical transplantation. (2011, background): “Secondary end points were: (1) to reduce the incidence and severity of bacterial, viral, and fungal infections and consequently transplantation-related mortality; and (2) to demonstrate that this strategy does not increase the incidence of posttransplantation leukemia relapse.”
HLA-haploidentical bone marrow transplantation for hematologic malignancies using nonmyeloablative conditioning and high-dose, posttransplantation cyclophosphamide. (2008, background): “For adult hematologic malignancies patients who lack suitably matched related or unrelated donors, encouraging outcomes have also been obtained with transplantation of HLAhaploidentical related stem cells after myeloablative conditioning [10,57] or with transplantation of 1 or 2 umbilical cord blood”
Donor natural killer cell allorecognition of missing self in haploidentical hematopoietic transplantation for acute myeloid leukemia: challenging its predictive value. (2006, background): “Donors who were classified as NK alloreactive against their recipients, termed NK alloreactive donors throughout, possessed (1) HLA class I KIR ligand(s) which were missing in the recipients, (2) KIR gene(s) for missing self recognition on recipient targets, and (3) alloreactive NK clones against re”