SarT, a Repressor of α-Hemolysin inStaphylococcus aureus

By K. Schmidt, A. Manna, S. Gill, A. Cheung. Infection and Immunity, 2001

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Autoinduction and signal transduction in the regulation of staphylococcal virulence (2003, background): “Moreover, in vitro analyses have revealed only inhibition by SarA (Chakrabarti and Misra, 2000), consistent with the suggestion by Arvidson and Tegmark (2001) that SarA is not ordinarily a transcriptional activator, and with the conclusion that SarA upregulates hla by downregulating the hla represso”
Bacterial quorum sensing: its role in virulence and possibilities for its control. (2012, background): “There is evidence for regulation by ArlRS, CcpA, CodY, Rsr, sB, SarA, SarR, SarT, SarU, SarX, SarZ, Rsr, and SrrA/SrrB (Cheung and Projan 1994; Schmidt et al. 2001; Yarwood et al. 2001; Manna and Cheung 2003, 2006a,b; Liang et al. 2005; Seidl et al. 2006; Majerczyk et al. 2008; Lauderdale et al.…”
The Genera Staphylococcus and Macrococcus (2006, background): “This finding suggests that sarA, contrary to the regulatory action of agr, induced α-hemolysin production by repressing sarT, a repressor of hla transcription (Schmidt et al., 2001).”