Mitochondrial dysfunction in cardiac disease: ischemia--reperfusion, aging, and heart failure.
By E. Lesnefsky, S. Moghaddas, Bernard Tandler, J. Kerner, C. Hoppel. Journal of Molecular and Cellular Cardiology, 2001
729 citations recorded by the source corpus.
Subfields: Molecular Biology, Pathology and Forensic Medicine
View Paper: https://www.semanticscholar.org/paper/f1cb8a4d1a128dd38bd753be7e7da0794fa9609e
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In Molecular Biology
Citations in the corpus
- Reperfusion injury and reactive oxygen species: The evolution of a concept (2015, background): “In addition, there are significant differences among species, tissues, cells, and even intracellular pools from which the mitochondria are derived [9,286,287].”
- Calcium, ATP, and ROS: a mitochondrial love-hate triangle. (2004, background): “Several other mitochondrial enzymes are inhibited in response to I/R or ROS exposure, including aconitase, -ketoglutarate dehydrogenase, and complexes III and IV (105, 161).”