Angiotensin II causes hypertension and cardiac hypertrophy through its receptors in the kidney
By S. Crowley, S. Gurley, M. Herrera, P. Ruiz, R. Griffiths, Anil P. Kumar, Hyung-Suk Kim, O. Smithies, T. Le, T. Coffman. Proceedings of the National Academy of Sciences of the United States of America, 2006
702 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Nutrition and Dietetics
View Paper: https://www.semanticscholar.org/paper/f031372b8b6698aa6bb3312da0b55415b1da0356
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- A. Ulloa-Aguirre
- J. Ito
- A. Nishiyama
- Per-Johan Jakobsson
- R. Morgenstern
- H. Kobori
- J. Janovick
- L. Navar
Citations in the corpus
- The Intrarenal Renin-Angiotensin System: From Physiology to the Pathobiology of Hypertension and Kidney Disease (2007, background): “Recently Crowley et al. (2006) have given an important insight on a crucial role of the intrarenal RAS by using a kidney cross-transplantation strategy to separate the actions of AT1 receptor pools in the kidney from those in systemic tissues (Crowley et al., 2006).”