Specific autoantibodies precede the symptoms of rheumatoid arthritis: a study of serial measurements in blood donors.

By M. Nielen, D. van Schaardenburg, H. Reesink, R. J. van de Stadt, I. E. van der Horst-Bruinsma, M. de Koning, M. R. Habibuw, Jan P. Vandenbroucke, B. Dijkmans. Arthritis & Rheumatism, 2004

1,966 citations recorded by the source corpus.

Subfields: Hepatology, Rheumatology

View Paper: https://www.semanticscholar.org/paper/eab3468db148bded5de4c624b1eb521606cc23d6

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Rheumatology

Citations in the corpus

Increased unrecognized coronary heart disease and sudden deaths in rheumatoid arthritis: a population-based cohort study. (2005, background): “Furthermore, recent studies demonstrated the presence of systemic inflammatory activity and serologic abnormalities several years prior to the onset of RA symptoms (37).”
Toward an integrated clinical, molecular and serological classification of inflammatory bowel disease: report of a Working Party of the 2005 Montreal World Congress of Gastroenterology. (2005, background): “1Department of Medicine, Mount Sinai Hospital IBD Centre, University of Toronto, Toronto, Ontario; 2Department of Gastroenterology, Western General Hospital, University of Edinburgh, Edinburgh; 3Gastroenterology Unit, University of Oxford, Gibson Laboratories, Radcliffe Infirmary, Oxford, United Kin”
An expansion of rare lineage intestinal microbes characterizes rheumatoid arthritis (2016, background): “Autoreactive T-cell responses and auto-antibody production leading up to the onset of RA occur much earlier than the clinical presentation of RA [34].”
Analysis of families in the multiple autoimmune disease genetics consortium (MADGC) collection: the PTPN22 620W allele associates with multiple autoimmune phenotypes. (2005, neutral): “Indeed , SLE, RA, T1D, and Hashimoto thyroiditis are all characterized by the frequent development of autoantibodies , often before the onset of clinical disease (Arbuckle et al. 2003; Strieder et al. 2003; Hoppu et al. 2004; Nielen et al. 2004).”