Type 2 diabetes across generations: from pathophysiology to prevention and management.
By Christopher J. Nolan, P. Damm, M. Prentki. The Lancet, 2011
920 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Genetics, Physiology
View Paper: https://www.semanticscholar.org/paper/e919853ac6fb022642b85b9e62ef260c40e4c03e
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Citations in the corpus
- Guidelines for the prevention of stroke in patients with stroke and transient ischemic attack: a guideline for healthcare professionals from the American Heart Association/American Stroke Association. (2014, background): “ily by excess calorie intake in people who are susceptible by virtue of inherited traits, age, and acquired behaviors.(54,112) In”
- The Pathophysiology of Gestational Diabetes Mellitus (2018, background): “This is an important distinction when considering potential treatments for GDM: attempts to directly reverse skeletal muscle insulin resistance, without reducing plasma glucose concentrations, could be detrimental [123].”
- Current hydrogel advances in physicochemical and biological response-driven biomedical application diversity (2021, background): “Islet cell delivery for diabetes therapy: Diabetes are primarily caused by the absolute or relative deficiency of insulin.(366,367) Pancreatic β-cell lines are usually responsible for regulating insulin production and secretion, especially for MIN6 cells.”
- AMPK, insulin resistance, and the metabolic syndrome. (2013, neutral): “, by increasing oxidative stress) is presently under debate (25, 26).”
- Wound repair and regeneration: Mechanisms, signaling, and translation (2014, neutral): “Currently, the prevalence of type 2 diabetes is 6.4% in the world population and is anticipated to increase to close to 8% in the year 2030 (10).”