Endoplasmic Reticulum Stress
By G. Bánhegyi, P. Baumeister, A. Benedetti, Dezheng Dong, Yong Fu, Amy S. Lee, Jianze Li, Changhui Mao, É. Margittai, Min Ni and 6 more. Annals of the New York Academy of Sciences, 2007
837 citations recorded by the source corpus.
Subfields: Cell Biology, Epidemiology, Surgery
View Paper: https://www.semanticscholar.org/paper/e90b0dfaf813b203859c66c5904259fec9d03b39
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Citations in the corpus
- A review of soil NO transformation: associated processes and possible physiological significance on organisms (2015, background): “…and Billiar, 1995; Lange et al., 2009; Pathogenesis of Type I diabetes due to NO induced islet cell death Overproduction of NO Endocrine system Oyadomari et al., 2002 Apoptosis due to eliminating Ca2+ from endoplasmic reticulum Overproduction of NO Pancreatic β- cells Oyadomari et al., 2001,…”
- ER stress-induced cell death mechanisms. (2013, background): “For example, high fat diet (HFD) is a well-known inducer of UPR and pathological ER stress signaling (reviewed in [94]).”
- ROS-mediated abiotic stress-induced programmed cell death in plants (2015, background): “In case these processes are impaired due to detrimental environmental factors, a specific phenomenon termed as “ER stress” can ensue, which is characterized by the accumulation of misfolded proteins (Cai et al., 2014).”
- Insulin resistance: Review of the underlying molecular mechanisms (2018, background): “Endoplasmic reticulum (ER) is a key organelle in eukaryotic cells, involved in synthesis, folding, packaging and transport of proteins, including insulin, by chaperone proteins, and glycosidase (Bánhegyi et al., 2007).”