ING tumor suppressor proteins are critical regulators of chromatin acetylation required for genome expression and perpetuation.

By Y. Doyon, Christelle Cayrou, M. Ullah, Anne-Julie Landry, Valérie Côté, W. Selleck, William Arbuthnot Sir Lane, Song Tan, Xiang-Jiao Yang, J. Côté. Molecules and Cells, 2006

691 citations recorded by the source corpus.

Subfields: Genetics, Molecular Biology

View Paper: https://www.semanticscholar.org/paper/e8336316ebebbf8256f2c3cef0d745b67d2e280a

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In Genetics

1. C. D. Allis
2. Dinshaw J. Patel
3. Alexander J. Ruthenburg
4. Haitao Li

In Molecular Biology

1. Alexander J. Ruthenburg
2. Dinshaw J. Patel
3. Haitao Li
4. P. Chi
5. G. Wang
6. C. D. Allis
7. Sean D. Taverna
8. D. Reinberg

Citations in the corpus

• The interplay of histone modifications – writers that read (2015, neutral): “The function of these accessory proteins remains unclear, although”
• Covalent histone modifications: miswritten, misinterpreted, and miserased in human cancers (2010, background): “2d ), whereas ING3, ING4 and ING5 recruit HATs to induce gene activatio”
• How chromatin-binding modules interpret histone modifications: lessons from professional pocket pickers (2007, background): “Similar structural parameters governing PHD module recognition are found in chromatin regulators, including those dedicated to activating or repressive activitie”