SK3 channel and mitochondrial ROS mediate NADPH oxidase-independent NETosis induced by calcium influx

By D. Douda, M. Khan, H. Grasemann, N. Palaniyar. Proceedings of the National Academy of Sciences of the United States of America, 2015

635 citations recorded by the source corpus.

Subfields: Genetics, Immunology, Pulmonary and Respiratory Medicine

View Paper: https://www.semanticscholar.org/paper/e7266da32353934243fe9d677328d6668b764321

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Genetics

1. V. Brinkmann
2. Arturo Zychlinsky

In Immunology

1. V. Brinkmann
2. Arturo Zychlinsky
3. P. Kubes
4. V. Papayannopoulos
5. N. Borregaard
6. A. Mócsai
7. B. Yipp
8. K. Martinod

In Pulmonary and Respiratory Medicine

1. Arturo Zychlinsky
2. V. Brinkmann

Citations in the corpus

• Diverse stimuli engage different neutrophil extracellular trap pathways (2017, background): “The role of ROS production in the generation of NETs is further confounded by recent work demonstrating that the mitochondria can also be a source of ROS in NETosis in response to calcium ionophores (Douda et al., 2015) or ribonucleoprotein-containing immune complexes (RNP ICs) (Lood et al., 2016).”
• Reactive oxygen species and mitochondria: A nexus of cellular homeostasis (2015, neutral): “2) [81].”