Cytokines in inflammatory bowel disease
By Markus F. Neurath. Nature reviews. Immunology, 2014
2,633 citations recorded by the source corpus.
Subfields: Genetics, Immunology
View Paper: https://www.semanticscholar.org/paper/e51f909af0e08407ef5690c4f5177ca6317b7bc7
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Citations in the corpus
- Mechanisms of Disease: Inflammatory Bowel Diseases. (2019, background): “Interestingly, UC-specific Th cells only show low levels of IL-4 production, which suggest they do not display all the features of classical Th2 cells (94, 95).”
- Single-Cell Analysis of Crohn’s Disease Lesions Identifies a Pathogenic Cellular Module Associated with Resistance to Anti-TNF Therapy (2019, background): “Infl. macs preferentially expressed neutrophilattracting chemokines (CXCL2, CXCL3, CXCL8) and cytokines essential for lymphocytes and stromal-cell activation like IL-23, IL-6, IL-1β, IL-1α, TNF, and OSM (Neurath, 2014; Powell et al., 2015; West et al., 2017), while activated DCs were enriched in ada”
- JAK–STAT Signaling as a Target for Inflammatory and Autoimmune Diseases: Current and Future Prospects (2017, background): “Moreover, anti-inflammatory cytokines such as IL-22 and IL-17A may have a protective role [104].”