NF-kappaB: a key role in inflammatory diseases.
By P. Tak, Gary S. Firestein. Journal of Clinical Investigation, 2001
1,410 citations recorded by the source corpus.
View Paper: https://www.semanticscholar.org/paper/e272a33863d41090eea16c8af61d8e787336277f
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Citations in the corpus
- NF-κB activation as a pathological mechanism of septic shock and inflammation (2006, unknown): “Activation of the NF- B pathway plays important role in the pathogenesis of rheumatoid arthritis and other forms of arthritis (234).”
- Targets of curcumin (2011, background): “Therefore, inhibition of pro-inflammatory cytokine production by regulation of transcriptional factors, such as NF-κB, is an potential strategy for controlling inflammatory responses [142,143].”
- Transcriptional regulation of collagenase (MMP-1, MMP-13) genes in arthritis: integration of complex signaling pathways for the recruitment of gene-specific transcription factors (2001, background): “Beyond the particularities of their receptors, however, IL-1 and TNF-α elicit a series of shared phosphorylation events within the cells that facilitate transcriptional induction of MMPs.”
- NF‐κB and the link between inflammation and cancer (2012, background): “NF-jB is considered by most to be central in not only promoting the inflammatory response but also in its resolution (150, 210, 211).”
- KLF2 Is a Novel Transcriptional Regulator of Endothelial Proinflammatory Activation (2004, background): “Previous studies have demonstrated that NF- B is critical for the activation of both the VCAM-1 and E-selectin promoters in response to inflammatory stimuli (44, 45, 6, 7, 8).”
- Trimethylamine N‐Oxide Promotes Vascular Inflammation Through Signaling of Mitogen‐Activated Protein Kinase and Nuclear Factor‐κB (2016, background): “marker for endothelial cell activation, especially in the pathological context of atherosclerosis.(23,24) To investigate”