Variability in individual responsiveness to clopidogrel: clinical implications, management, and future perspectives.
By D. Angiolillo, A. Fernández-Ortiz, E. Bernardo, F. Alfonso, C. Macaya, T. Bass, M. Costa. Journal of the American College of Cardiology, 2007
1,016 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Surgery
View Paper: https://www.semanticscholar.org/paper/e25cbfeed4c0f2f2eca7e8d851d80e0d3a57eae9
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
- U. Tantry
- T. Pollin
- Coleen Damcott
- J. O’Connell
- A. Parsa
- N. Faraday
- K. Ryan
- Braxton D. Mitchell
Citations in the corpus
- Standard- vs high-dose clopidogrel based on platelet function testing after percutaneous coronary intervention: the GRAVITAS randomized trial. (2011, neutral): “BMI, median (range) 31 (15-66) 31 (15-60) .”
- Association of cytochrome P450 2C19 genotype with the antiplatelet effect and clinical efficacy of clopidogrel therapy. (2009, background): “Variability in clopidogrel response is well established.(6-8) Patients treated with clopidogrel who demonstrate higher ex vivo platelet reactivity are at increased risk of ischemic events.”