Metabolic effects of fructose and the worldwide increase in obesity.
By L. Tappy, K. Lê. Physiological Reviews, 2010
1,216 citations recorded by the source corpus.
Subfields: Endocrinology, Diabetes and Metabolism, Epidemiology, Physiology
View Paper: https://www.semanticscholar.org/paper/dc54a49764287d60fb1a3c57be840e10bbf9a3e3
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In Endocrinology, Diabetes and Metabolism
In Epidemiology
In Physiology
Citations in the corpus
- Pathophysiology of human visceral obesity: an update. (2013, background): “Overall, available studies demonstrate that fructose consumption increases fasting triglyceride and glucose levels, stimulates deposition of triglycerides in nonadipose tissues (ectopic fat), deteriorates glucose and insulin responses to an oral sucrose challenge, and leads to hepatic insulin resist”
- Palmitic Acid: Physiological Role, Metabolism and Nutritional Implications (2017, background): “Unlike glucose, fructose, being taken up almost totally by the liver (Tappy and Le, 2010), cannot be used for glycogen biosynthesis and is promptly converted to glyceraldehyde-3-phosphate, providing a substrate for DNL.”
- Global gender disparities in obesity: a review. (2012, background): “Although increased SSB intake is associated with the increased prevalence of obesity in the United States among men and women, dairy intake may differentially affect sex-specific weight gain (33,34).”