Spotty Calcification Typifies the Culprit Plaque in Patients With Acute Myocardial Infarction: An Intravascular Ultrasound Study

By S. Ehara, Yoshiki Kobayashi, M. Yoshiyama, K. Shimada, Y. Shimada, D. Fukuda, Yasuhiro Nakamura, H. Yamashita, H. Yamagishi, K. Takeuchi and 5 more. Circulation, 2004

697 citations recorded by the source corpus.

Subfields: Cardiology and Cardiovascular Medicine, Radiology, Nuclear Medicine and Imaging, Surgery

View Paper: https://www.semanticscholar.org/paper/da4a091b5abc653a1e45b91c85428d50c0e8e86f

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Radiology, Nuclear Medicine and Imaging

1. Erling Falk
2. J. Bentzon
3. Renu Virmani

In Surgery

1. Erling Falk
2. J. Bentzon
3. A. Finn
4. F. Otsuka
5. Renu Virmani

Citations in the corpus

• Mechanisms of plaque formation and rupture. (2014, background): “Furthermore, culprit lesions for ACS are generally less calcified than plaques responsible for stable angina, and the pattern of plaque calcification also differs.(80,142) These features are not independently associated with plaque rupture, however, and if there is a causal link, it is probably thro”
• Update on acute coronary syndromes: the pathologists' view. (2013, background): “lower-risk plaques.(111) –113 The total amount of calcification—the CAC score—is a marker of plaque burden (and thus a marker of cardiovascular risk) rather than a marker of risk conferred by the individual plaque.”