Immunity to tuberculosis.

By R. North, Y. Jung. Annual Review of Immunology, 2003

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Foamy macrophages and the progression of the human TB granuloma (2009, background): “At this time, although the tissue response is relatively unstructure”
Mycobacterium tuberculosis: Success through dormancy (2012, background): “Macrophage activation via cytokines, notably, interferon-gamma (IFN-c), for instance, allows these host cells to control their intracellular predators (Cooper et al., 1993; North & Jung, 2004).”
The Role of the Granuloma in Expansion and Dissemination of Early Tuberculous Infection (2009, background): “…thought to form only after initiation of adaptive immunity (Saunders and Cooper, 2000), and in animal models of tuberculosis , bacterial growth is rapid for the first 2 weeks of infection and plateaus coincident with the development of adaptive immunity (North and Jung, 2004; Swaim et al., 2006).”
The immune response in tuberculosis. (2013, background): “tuberculosis infection and may have cleared infection through an effective innate immune response (13), although either case is difficult to prove in humans.”
Human T cell epitopes of Mycobacterium tuberculosis are evolutionarily hyperconserved (2010, neutral): “By contrast, MTBC causes chronic and often lifelong infections, and adaptive immunity is usually unable to completely clear the infection 18 .”