Pathophysiology of ischaemia–reperfusion injury

By D. Carden, D. Granger. Journal of Pathology, 2000

1,101 citations recorded by the source corpus.

Subfields: Emergency Medicine, Pathology and Forensic Medicine, Physiology

View Paper: https://www.semanticscholar.org/paper/d9dcd29a28fd39bd714309ba0ff6c1be5330157b

How researchers see this paper

The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.

In Emergency Medicine

1. M. Krenz
2. Ronald J. Korthuis
3. Christopher P. Baines
4. Theodore J Kalogeris

In Pathology and Forensic Medicine

1. M. Krenz
2. Ronald J. Korthuis
3. C. Iadecola
4. Christopher P. Baines
5. Theodore J Kalogeris

In Physiology

1. M. Krenz
2. Ronald J. Korthuis
3. J. Anrather
4. C. Iadecola
5. Christopher P. Baines
6. Theodore J Kalogeris
7. Berislav Zlokovic

Citations in the corpus

• The immunology of stroke: from mechanisms to translation (2011, background): “The inflammatory process begins in the intravascular compartment immediately after arterial occlusion, when the ensuing hypoxia, changes in shear stress and production of reactive oxygen species (ROS) trigger the coagulation cascade, and lead to activation of complement, platelet and endothelial cel”
• Cell Biology of Ischemia/Reperfusion Injury (2012, background): “In this regard, the lungs are especially vulnerable, particularly after I/R of the gut and/or liver (Carden and Granger, 2000; He et al., 2011a,b; Sorkine et al., 1997), as they are the first major capillary bed exposed to postischemic blood.”
• Ischemia/Reperfusion (2016, positive): “to the deleterious effects of distant organ ischemia, particularly after bowel and/or liver I/R (114, 331, 733).”