Mechanisms of Resistance to Bispyribac-Sodium in an Echinochloa phyllopogon Accession

By A. Fischer, D. Bayer, Michael D. Carriere, Comfort M. Ateh, Kyu-Ock Yim. 2000

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Cross-resistance to bispyribac-sodium and Bensulfuron-methyl in Echinochloa phyllopogon and Cyperus difformis (2002, background): “oryzoides and the new herbicide bispyribac-sodium that has not yet been used [7,11].”
Basis for Herbicide Resistance in Canadian Populations of Wild Oat (Avena fatua) (2012, background): “The addition of malathion at this dose before treatment of the ALS inhibitors bispyribac (Fischer et al. 2000) and penoxsulam (Yasuor et al. 2009) to ALS inhibitor-resistant rice barnyardgrass [Echinochloa phyllopogon (Stapf) Kosso-Pol.] was also found to enhance herbicide phytotoxicity, attributed…”
Plant cytochrome P450-mediated herbicide metabolism (2006, background): “For instance, Fischer et al. (2000) suggested that watergrass resistance to bispyribac-sodium, a pyrimidinyloxybenzoate herbicide commercially unavailable at the time of the study, emerged due to enhanced detoxification by a P450 that was selected by the continuous use of molinate.”
Herbicide Metabolism: Crop Selectivity, Bioactivation, Weed Resistance, and Regulation (2019, methodology): “Greenhouse experiments conducted on the plants collected from fields in 1997 revealed that the plants exhibited resistance to fenoxaprop-ethyl, molinate, thiobencarb, and bispyribac-sodium, a herbicide that was not released yet (Fischer et al. 2000a; Osuna et al. 2002).”