Regulation of contraction in striated muscle.
By A. M. Gordon, E. Homsher, M. Regnier. Physiological Reviews, 2000
1,682 citations recorded by the source corpus.
Subfields: Cardiology and Cardiovascular Medicine, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/d638576ff7c07dc1b7770fd9e6606eb933e7ac00
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In Cardiology and Cardiovascular Medicine
In Molecular Biology
Citations in the corpus
- Fiber types in mammalian skeletal muscles. (2011, background): “Whereas the regulatory mechanism itself has been the subject of a specific review (276), the focus of the present review is restricted on the structural and functional diversity among muscle fiber types.”
- Skeletal muscle fatigue: cellular mechanisms. (2008, background): “First, increased Pi might reduce the myoplasmic Ca(2) buffering by decreased binding of Ca(2) to troponin C due to a reduction in strong cross-bridge attachment (314), but this mechanism appears to be of limited importance in skeletal muscle (192).”
- Physiology and pathophysiology of carnosine. (2013, background): “The excitation-contraction coupling in skeletal muscle cells is a complexly regulated chain of events in which calcium is released from the sarcoplasmic reticulum (SR) through ryanodine receptors (RyR), the released calcium will bind to troponin C to allow cross-bridge formation and force production”