Mechanisms of hepatic fibrogenesis.
By S. Friedman. Gastroenterology, 2008
1,575 citations recorded by the source corpus.
Subfields: Epidemiology, Genetics, Hepatology
View Paper: https://www.semanticscholar.org/paper/d5b5fc3421db05aadd14f3593ef4d82bd40cf778
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Citations in the corpus
- Liver Fibrosis: Mechanistic Concepts and Therapeutic Perspectives (2020, background): “In addition, activated cells express high levels of alpha Smooth Muscle Actin (α-SMA) and Tissue Inhibitor of Metalloproteinase 1 (TIMP1) which contribute to the changes from a adipocytic phenotype to a pro-fibrogenic and inflammatory phenotype [54,55].”
- TGF-β/SMAD Pathway and Its Regulation in Hepatic Fibrosis (2016, background): “In patients with HF, increased concentrations of TGF-β1 correlate with the severity of hepatic fibrosis, suggesting a link between TGF-β expression and progressive liver disease (Friedman 2008; Gressner and Weiskirchen 2006; Lee and Friedman 2011).”
- Kupffer Cells in the Liver (2013, neutral): “While MMPs can actually contribute to fibrosis during the progression stage, they can also promote the removal of extracellular matrix during the resolution of fibrosis (30).”