Biosynthesis of plant phenolic compounds in elevated atmospheric CO2
By S. Hartley, Clive G. Jones, G. Couper, T. Jones. 2000
129 citations recorded by the source corpus.
Subfields: Plant Science
View Paper: https://www.semanticscholar.org/paper/d47e6393485c8f007aa58b06701a033e6cd7b8cd
How researchers see this paper
The researchers whose trusted networks rank it highest, strongest advocate first. Every researcher gets a different order.
In Plant Science
- Andrew J. McElrone
- D. Ballhorn
- S. Kautz
- S. Chakraborty
- Jason Hamilton
- R. B. Jackson
- Rob Norton
- Alistair Rogers
Citations in the corpus
- Genetic and environmental interactions determine plant defences against herbivores (2011, background): “In the last decades, research has demonstrated how plants can actively up-regulate a wide array of defensive traits when attacked by herbivores or pathogens or being subjected to abiotic stress factors (e.g. Hartley et al. 2001; Ballhorn, Heil & Lieberei 2006; Saadatmand et al. 2008).”
- Elevated CO2 reduces disease incidence and severity of a red maple fungal pathogen via changes in host physiology and leaf chemistry (2005, background): “Altered leaf nutritional quality caused by reduced N and increased defensive chemistry under elevated CO2 is a likely mechanism functioning in many pathosystems, given the frequent occurrence of these responses across numerous plant species (Hartley et al., 2000).”
- Anthropogenic Changes in Tropospheric Composition Increase Susceptibility of Soybean to Insect Herbivory (2005, background): “Growth under elevated CO2 alters many aspects of leaf chemistry potentially affecting herbivory, including secondary plant compounds (Bezemer and Jones 1998, Hartley et al. 2000 ).”
- Production and fitness of Fusarium pseudograminearum inoculum at elevated carbon dioxide in FACE (2010, background): “These studies point to changes in anatomy, morphology and physiology to influence disease resistance in plants (Hibberd et al., 1996; Hartley et al., 2000; Karnosky et al., 2002; Pangga et al., 2004; Kobayashi et al., 2006; Matros et al., 2006); changes in pathogen life cycle (Chakraborty et al.,…”