CNS plasticity and assessment of forelimb sensorimotor outcome in unilateral rat models of stroke, cortical ablation, parkinsonism and spinal cord injury.

By T. Schallert, Sheila M. Fleming, J. Leasure, Jennifer L. Tillerson, S. T. Bland. Neuropharmacology, 2000

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Animal models of Parkinson's disease: a source of novel treatments and clues to the cause of the disease (2011, methodology): “Deficits in use of the contralateral forelimb (reflective of akinesia) are also found post lesion in the vibrissae-elicited forelimb placement test, which assesses the ability of an animal to place its forelimb on a bench edge in response to detection of vibrissae movements caused by contact with sa”
Pharmacological validation of behavioural measures of akinesia and dyskinesia in a rat model of Parkinson's disease (2002, neutral): “Injections of bromocriptine, L -DOPA and saline were made 120, 45 and 20 min, respectively, before the recordings started.”
Regulatory T cells are key cerebroprotective immunomodulators in acute experimental stroke (2009, methodology): “Functional neurological deficit was assessed by the forelimb use asymmetry tes”
Selenium Drives a Transcriptional Adaptive Program to Block Ferroptosis and Treat Stroke. (2019, neutral): “Behavioral analysis The corner task assessed the integrated sensorimotor function in both stimulation of vibrissae (sensory neglect) and rearing (motor response; Schallert et al., 2000; Karuppagounder et al., 2016).”