Endocannabinoid-mediated control of synaptic transmission.

By Masanobu Kano, T. Ohno-Shosaku, Y. Hashimotodani, M. Uchigashima, Masahiko Watanabe. Physiological Reviews, 2009

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Distinct coincidence detectors govern the corticostriatal spike timing‐dependent plasticity (2010, background): “As previously observed in endocannabinoid-mediated short-term plasticity, 2-AG signalling depends on activation of mGluR5 or M1Rs, which are Gq/11 protein-coupled receptors to PLCβ (Chevaleyre et al. 2006; Kano et al. 2009).”
Cannabinoid Receptors and the Endocannabinoid System: Signaling and Function in the Central Nervous System (2018, background): “Several models have been proposed to elucidate the transport of AEA: simple diffusion driven by concentration gradients generated from enzymatic degradation, endocytosis involving caveolae/lipid rafts, through certain carrier proteins like fatty acid binding proteins and heat shock protein 70 [9].”
Neurobiological Interactions Between Stress and the Endocannabinoid System (2016, background): “…on GABAergic, glutamatergic, serotonergic, noradrenergic, and dopaminergic terminals (Azad et al, 2008; Haring et al, 2007; Hermann et al, 2002; Kano et al, 2009; Morozov et al, 2009; Oropeza et al, 2007), but given the relative abundance of excitatory and inhibitory neurons in the brain, and…”