A synaptic trek to autism.
By Thomas Bourgeron. Current Opinion in Neurobiology, 2009
659 citations recorded by the source corpus.
Subfields: Genetics, Molecular Biology
View Paper: https://www.semanticscholar.org/paper/ccc34c0ad77ad618500153f1129d2a1ecf588a00
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Citations in the corpus
- Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits (2014, positive): “Dysregulated mTOR signaling has been identified in autism, fragile X syndrome, tuberous sclerosis, neurofibromatosis, and PTEN-mediated macroencephaly (Peça and Feng, 2012; Bourgeron, 2009), each of which features altered dendritic spine densities.”
- Is eye contact the key to the social brain? (2010, background): “[aPMN] Bourgeron, T. (2009) A synaptic trek to autism.”
- Dendritic spine pathology in neuropsychiatric disorders (2011, background): “and Shank3 may complex with and signal downstream of NLGN”
- Shank3 mutant mice display autistic-like behaviours and striatal dysfunction (2011, background): “isolation from astrocytes and blood vessels and (4) morphologically, by the presence of high number of spines and relatively short neuronal arborizations as”